Diabetes Treatment In Ayurveda
Diabetes has been known since the first century…..when a Greek physician, A medical condition in which the body can’t produce enough insulin to process the glucose in the blood is known as diabetes.
Due to these daily habits, every other
person is having diabetes or bee problem And within a short time there is also
control nowadays. Now children are also seen in the grip of this disease The
word diabetes was first recorded in 1425, and in 1675, the Greek
word Mellitus, meaning “like honey,” was added, to reflect the
sweet smell and taste of the patient’s urine. An unrelated and rare disorder
similarly in Ayurveda a condition in which a person passes honey-like (sweet)
urine is called Madhumeha(Hyperglycemia)Reduced insulin production and
decreased insulin sensitivity are the contributing factors for hyperglycemia.
Today we are going to tell Ayurvedic treatment
related to this disease.
Of these, there are ways that it is said that
using it causes diabetes to get into sugar very quickly. Sanjivani
Ayurvedashram doctor says Traditionally for diabetes control These two
tips to be used Scientifically proven to be very valuable Today we will talk
about such tunes to those who are traditional today we will talk about such
tunes to those who are traditional Remedies for diabetic patients Under this,
the treatment of
diabetes is going on There's no need to shut it down From members to
control diabetes Traditionally they are being used Sanjivani
Ayurvedic is going to tell you about these remedies, for which you
will not need to stop any medicine to use them.
Traditionally, it is still being used, but
only a few of which are predominantly tribal. Okay, I am going to tell you
about the prescriptions of the future, use it carefully, and use your medicines
which are indigenous Sanjivani Ayurvedashram. A medical
condition in which the body can’t produce enough insulin to process the glucose
in the blood is known as diabetes
- Chew a spoonful of flaxseed seeds, eat them well and
drink two glasses of water, do it daily on an empty stomach in the morning
and before bed at night.
Classification
·
Diabetes is classified on the basis of its etiology
·
Type-I DM - Due to 'Beta' cell destruction, usually leading to
absolute insulin deficiency.
·
Type-II DM - Due to a variable degree of Insulin resistance
Impaired Insulin secretion
·
Glucose intolerance and insulin resistance which generally
increases with the advancement of age.
·
Other specific types of Diabetes due to Impaired Beta-cell
function due to genetic defects Impaired Insulin action due to genetic defects
·
Diseases of pancreas
·
Endocrinopathies
·
Drugs (chemically induced)
·
Infections-Like congenital rubella, cytomegalovirus, etc.
Risk factors
·
Family history
·
Obesity (BMI > 27kg/ m2)
·
Age > 45 years
·
Hypertension (B.P. > 140/ 90 mm of Hg)
·
HDL < 35mg/ dl and/ or triglycerides levels > 250mg/ dl
· Habitual physical inactivity
Clinical features
·
Increased frequency of urine(Polyuria)
·
Increased appetite (Polyphagia)
·
Excessive thirst (polydipsia)
·
Turbidity in urine
·
Debility/ tiredness
·
Weight loss
·
Non-healing ulcer
·
Visual disturbances
·
Inflammation of glans penis
· The presentation of symptoms in elderly patients with diabetes
may be significantly different from the classic triad of polyuria, polydipsia,
polyphagia, and weight loss. They may present with fatigue, anorexia, failure
to thrive, loss of motivation, difficulty in concentration, and urinary
incontinence.
Complications
In the later stage of diabetes mellitus, the following complications may appear
Burning sensation (neuropathy) over the palmar and plantar
region
·
Boils and carbuncles
·
Gangrene
·
General debility
·
Retinopathy
·
Renal tissue damage (nephropathy)
·
Cardiovascular diseases
·
Disease-Specific
i. Measurement of the plasma glucose level
a. Random blood sugar (RBS)
b. Fasting blood sugar (FBS)
. Postprandial blood sugar (PPBS)
ii. Urine routine and microscopic
iii. Glycosylated haemoglobin (HbA1c)
. Lipid Profile
Other related
Investigations
i. Blood urea and serum creatinine
ii. E.C.G.
iii. Fundus examination
iv. Serum electrolytes
Diagnosis
The diagnosis is made by corroborating
symptoms and plasma glucose levels
·
Fasting plasma glucose > 126 mg/dl after overnight fasting
(or)
·
Random plasma glucose > 200 mg/dl (or)
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